“Aluminum As a Cause of Senile Dementia”, 1982-05 (; similar):
The evidence reviewed shows that senile dementia may be similar in origin to Alzheimer’s disease and to dialysis encephalopathy. There is general agreement that aluminum, once attached to the chromatin in a neuron, cannot be dislodged by any means available to the organism. Yet the presence of aluminum in serum shows that at least some trace will always be able to pass biological barriers and ultimately reach critical neuronal chromatin.
Alfrey shows that the aluminum content of heart and brain remains relatively low until the bone content nears a saturation point, after which aluminum deposition in heart and brain accelerates (Figure 1).16 The data on aluminum content of the human aorta by Zinsser, Bjorksten, et al indicate that aluminum content peaks from age 40 to 50 years, and declines moderately thereafter.17 Thus, it is possible that persons who have the highest body level of aluminum may not survive for 5 years, but more data are needed to prove this theory.
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